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Sarah M Kamel
Sarah M Kamel
PhD candidate, Hubrecht Institute
Adresse e-mail validée de hubrecht.eu
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Année
MUSCLEMOTION: a versatile open software tool to quantify cardiomyocyte and cardiac muscle contraction in vitro and in vivo
L Sala, BJ Van Meer, LGJ Tertoolen, J Bakkers, M Bellin, RP Davis, ...
Circulation research 122 (3), e5-e16, 2018
2752018
Effective CRISPR/Cas9-based nucleotide editing in zebrafish to model human genetic cardiovascular disorders
F Tessadori, HI Roessler, SMC Savelberg, S Chocron, SM Kamel, ...
Disease models & mechanisms 11 (10), dmm035469, 2018
812018
ABCC9-related Intellectual disability Myopathy Syndrome is a KATP channelopathy with loss-of-function mutations in ABCC9
MF Smeland, C McClenaghan, HI Roessler, S Savelberg, GÅM Hansen, ...
Nature communications 10 (1), 4457, 2019
372019
Frontline Science: Antagonism between regular and atypical Cxcr3 receptors regulates macrophage migration during infection and injury in zebrafish
F Sommer, V Torraca, SM Kamel, A Lombardi, AH Meijer
Journal of leukocyte biology 107 (2), 185-203, 2020
362020
Istaroxime treatment ameliorates calcium dysregulation in a zebrafish model of phospholamban R14del cardiomyopathy
SM Kamel, CJM van Opbergen, CD Koopman, AO Verkerk, BJD Boukens, ...
Nature communications 12 (1), 7151, 2021
182021
Inflammatory response in hematopoietic stem and progenitor cells triggered by activating SHP2 mutations evokes blood defects
M Solman, S Blokzijl-Franke, F Piques, C Yan, Q Yang, M Strullu, ...
Elife 11, e73040, 2022
122022
Loss of sdhb in zebrafish larvae recapitulates human paraganglioma characteristics
M Dona, S Waaijers, S Richter, G Eisenhofer, J Korving, SM Kamel, ...
Endocrine-Related Cancer 28 (1), 65-77, 2021
112021
Effective teaching of science in an undergraduate course; knowledge, discipline and dedication yield scientists
A Amleh, M Abou El-Soud, S Kamel
Procedia-Social and Behavioral Sciences 9, 890-902, 2010
112010
A Heterozygous Mutation in Cardiac Troponin T Promotes Ca2+ Dysregulation and Adult Cardiomyopathy in Zebrafish
SM Kamel, CD Koopman, F Kruse, S Willekers, S Chocron, J Bakkers
Journal of Cardiovascular Development and Disease 8 (4), 46, 2021
102021
Knockdown of COBRA1 decreases the proliferation and migration of hepatocellular carcinoma cells
E El Zeneini, S Kamel, M El-Meteini, A Amleh
Oncology reports 37 (3), 1896-1906, 2017
92017
A functional genomics screen identifying blood cell development genes in Drosophila by undergraduates participating in a course-based research experience
CJ Evans, JM Olson, BC Mondal, P Kandimalla, A Abbasi, ...
G3 11 (1), jkaa028, 2021
72021
Effective CRISPR/Cas9-based nucleotide editing in zebrafish to model human genetic cardiovascular disorders. Dis Model Mech 11 (10)
F Tessadori, HI Roessler, SMC Savelberg, S Chocron, SM Kamel, ...
52018
The zebrafish cohesin protein Sgo1 is required for cardiac function and eye development
SM Kamel, S Broekman, F Tessadori, E van Wijk, J Bakkers
Developmental Dynamics 251 (8), 1357-1367, 2022
32022
Correlating the expression levels of COBRA1 (co-factor of BRCA1) with hepatocellular carcinoma development in Egyptian patients
S Kamel
22013
Versatile open software to quantify cardiomyocyte and cardiac muscle contraction in vitro and in vivo
L Sala, BJ Van Meer, LGJ Tertoolen, J Bakkers, M Bellin, RP Davis, ...
bioRxiv, 160754, 2017
12017
CRISPR/Cas9 mutagenesis of zebrafish Cxcr3. 3 suggests opposing functions of atypical and canonical Cxcr3 paralogues on mycobacterial infection control
V Torraca, F Sommer, S Kamel, NA Otto, AH Meijer
Exploitation of host chemokine signalling by pathogenic mycobacteria 122, 0
1
Modeling human cardiac genetic diseases in the zebrafish: Focus on cardiomyopathies and cohesinopathies
S Kamel
2021
Antagonism between regular and atypical Cxcr3 receptors regulates macrophage migration during infection and injury in zebrafish
F Sommer, V Torraca, S Kamel, A Lombardi, AH Meijer
bioRxiv, 719526, 2019
2019
2Antagonism between regular and atypical Cxcr3 receptors
F Sommer, V Torraca, S Kamel, AH Meijer
Chemokine signaling mechanisms underlying inflammation and infection control …, 0
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